Cartilla Nacional de Vacu­nación

Las dosis son seguras: SSa

La Prensa
21 de febrero de 2011

Mario Mar tí nez

La Prensa
21 de febrero de 2011

Ma rio Mar tí nez

El ti tu lar de la Se cre ta ría de Sa lud, Jo sé An gel Cór do va Vi lla lo bos, ase gu ró que to das las va cu nas (ca­si 13.000,000) que se apli ca rán du­ran te la Se ma na Na cio nal de Va cu­na ción, no re pre sen tan nin gún ries­go.

Al acom pa ñar al Pre si den te Fe li pe Cal de rón en el arran que de la Se­ma na Na cio nal de Sa lud 2011 y de la inau gu ra ción del La bo ra to rio Na­cio nal de Va cu nas de la Co mi sión y Con trol Ana lí ti co de Co ber tu ra de la Co mi sión Fe de ral pa ra la Pro tec ción de Ries gos Sa ni ta rios (Co fe pris), in di có que és te ayu da­rá a for ta le cer el tra ba jo pa ra brin dar el res pal do ana lí ti co que co rro bo ra que to das las va cu­nas que son apli ca das, tan to en el sec tor pú bli co, co mo en el pri va do, no re pre sen ten ries go al gu no pa ra la po bla ción.

In di có que con lo an te rior, se lo gra rá in cre men tar la ca pa ci dad ana lí ti ca en un 30 por cien to, pa ra con tar con la ca pa ci dad de lle var a ca bo más de 5,000 aná li sis adi cio na les al año pa ra las va cu nas.

Su bra yó que aña de a su ca pa ci dad, un nue vo La bo ra to rio Na cio nal de Con trol de Ca li dad de Ni vel Bio se gu ri dad BSL-2 Plus, de acuer do con los es tán da res in ter na cio na les, y con ca pa ci­dad de in ter ve nir en si tua cio nes de emer gen cia bio ló gi ca o te rro ris ta.

Sin ex po ner, di jo, a los tra ba ja do res, o al me dio am bien te, a la di se mi na ción de agen tes bio­ló gi cos con pa to ge ni ci dad des co no ci da, lo que re pre sen ta, abun dó, una for ta le za pa ra la Se­cre ta ría de Sa lud (SSa).

Ex pli có que du ran te la Se ma na Na cio nal de Sa lud se ofre ce rán cua tro ser vi cios de aten ción bá si ca: va cu na ción a la po bla ción infantil prees co lar, es co lar y a las mu je res de 12 a 44 años de edad; em ba ra za das pa ra lo grar el con trol, eli mi na ción o erra di ca ción de las en fer me da des que se pue den evi tar con la apli ca ción de va cu nas.

Así co mo la di fu sión de la in for ma ción so bre pre ven ción del té ta nos neo na tal y no neo na tal; la dis tri bu ción de áci do fó li co a las mu je res en edad re pro duc ti va, pa ra evi tar que sus hi jos naz­can con al gu nas mal for ma cio nes con gé ni tas. Leer más http://www.oem.com.mx/laprensa/notas/n1975707.htm

Cartilla Nacional de Salud




Desarrolla México vacunas contra adicciones, dice Calderón

MÉXICO, D.F.- "Es algo que no se ha logrado en el mundo, es cierto, pero que investigadores mexicanos están realizando aquí", dijo el mandatario.


El secretario de Salud, José Ángel Córdova y el presidente Felipe Calderón durante la nauguración de la Unidad de Investigación Biopsicosocial en Adicciones. (Notimex)
  • El secretario de Salud, José Ángel Córdova y el presidente Felipe Calderón durante la nauguración de la Unidad de Investigación Biopsicosocial en Adicciones. (Notimex)
MÉXICO, D.F.- El presidente Felipe Calderón anunció la investigación de una vacuna contra la cocaína y dijo confiar en que tarde o temprano se desarrollen también contra la heroína, nicotina y metanfetaminas e incluso una pentavalente.

"Algo que no se ha logrado en el mundo, es cierto, pero que investigadores mexicanos están realizando aquí", dijo el mandatario al inaugurar el Centro de Investigación Biopsicosocial en Adicciones.

"Sé que el desarrollo de la vacuna tomará meses, años; tomará muchos recursos, hay que meterle recursos. Volveremos a romper el cochinito, ya no va a alcanzar el de Salud, pero vamos a tener que romper otros también, para que podamos desarrollarlo", dijo.

Leer más http://www.sipse.com/noticias/83935-desarrolla-mexico-vacunas-contra-adicciones-dice-calderon.html
APPLAC México a la vanguardia en el Síndrome de Post Polio

The Polio Crusade

THE POLIO CRUSADE IN AMERICAN EXPERIENCE A GOOD VIDEO THE STORY OF THE POLIO CRUSADE pays tribute to a time when Americans banded together to conquer a terrible disease. The medical breakthrough saved countless lives and had a pervasive impact on American philanthropy that ... Continue reading..http://www.pbs.org/wgbh/americanexperience/polio/

Erradicación de La poliomielitis

Polio Tricisilla Adaptada

March Of Dimes Polio History

Dr. Bruno




A 41-year-old man developed an acute illness at the age of 9 months during which, following a viral illness with headache, he developed severe weakness and wasting of the limbs of the left side. After several months he began to recover, such that he was able to walk at the age of 2 years and later was able to run, although he was never very good at sports. He had stable function until the age of 18 when he began to notice greater than usual difficulty lifting heavy objects. By the age of 25 he was noticing progressive difficulty walking due to weakness of both legs, and he noticed that the right calf had become larger. The symptoms became more noticeable over the course of the next 10 years and ultimately both upper as well as both lower limbs had become noticeably weaker.

On examination there was wasting of the muscles of upper and lower limbs on the left, and massively hypertrophied gastrocnemius, soleus and tensor fascia late on the right. The calf circumference on the right exceeded that on the left by 10 cm (figure1). The right shoulder girdle, triceps, thenar eminence and small muscles of the hand were wasted and there was winging of both scapulae. The right quadriceps was also wasted. The wasted muscles were also weak but the hypertrophied right ankle plantar flexors had normal power. The tendon reflexes were absent in the lower limbs and present in the upper limbs, although the right triceps was reduced. The remainder of the examination was normal.

Figure 1

The patient's legs, showing massive enlargement of the right calf and wasting on the left


What is that nature of the acute illness in infancy?
What is the nature of the subsequent deterioration?
What investigations should be performed?
What is the differential diagnosis of the cause of the progressive calf hypertrophy?



An acute paralytic illness which follows symptoms of a viral infection with or without signs of meningitis is typical of poliomyelitis. Usually caused by one of the three polio viruses, it may also occur following vaccination and following infections with other enteroviruses.1 Other disorders which would cause a similar syndrome but with upper motor neurone signs would include acute vascular lesions, meningoencephalitis and acute disseminated encephalomyelitis.


A progressive functional deterioration many years after paralytic poliomyelitis is well known, although its pathogenesis is not fully understood.2 It is a diagnosis of exclusion; a careful search for alternative causes, for example, orthopaedic deformities such as osteoarthritis or worsening scoliosis, superimposed neurological disorders such as entrapment neuropathies or coincidental muscle disease or neuropathy, and general medical causes such as respiratory complications and endocrinopathies.3


Investigations revealed normal blood count and erythrocyte sedimentation rate and normal biochemistry apart from a raised creatine kinase at 330 IU/l (normal range 60–120 IU/l), which is commonly seen in cases of ongoing denervation. Electromyography showed evidence of denervation in the right APB and FDI with polyphasic motor units and complex repetitive discharges, no spontaneous activity in the left calf and large polyphasic units in the right calf consistent with chronic partial denervation. Motor and sensory conduction velocities were normal. A lumbar myelogram was normal. Magnetic resonance imaging (MRI) scan of the calves is shown in figure2.

Figure 2

Axial T1 weighted MRI scan (TR 588 ms, TE 15 ms) of the calves, showing gross muscle atrophy and replacement by adipose tissue on the left, and hypertrophy of the muscles on the right, with only minor adipose tissue deposition


The differential diagnosis of the progressive calf hypertrophy is given in the box.

Causes of calf muscle hypertrophy

Chronic partial denervation

  • radiculopathy

  • peripheral neuropathy

  • hereditary motor and sensory neuropathy

  • spinal muscular atrophy

  • following paralytic poliomyelitis

    Neuromyotonia and myokymia

  • Isaac's syndrome

  • generalised myokymia

  • neurotonia

  • continuous muscle fibre activity due to: chronic inflammatory demyelinating polyradiculopathy, Guillain Barre syndrome, myasthenia gravis, thymoma, thyrotoxicosis, thyroiditis

    Muscular dystrophies



  • tumours

  • amyloidosis

  • cysticercosis

    Link here