Apr 27, 2017

If You Have Heart Problems Or High Blood Pressure Avoid These 14 Foods At All Costs





If You Have Heart Problems Or High Blood Pressure Avoid These 14 Foods At All Costs
The fact that nutrition is one of the main causes of coronary diseases shows that you should take care of your diet and be very careful if you want to maintain your health at optimal level.
There are some foods which can be harmful to your heart, have a negative effect on the blood circulation and cause many other serious health problems.
So, today we will present you 14 foods which can damage your health and you should avoid them at all cost if you want to protect yourself!
-Table salt
The excessive intake of salt can lead to water retention and impaired blood circulation, and increase the risk of kidney, heart, and brain problems.
Pizza
The Grocery Manufacturers Association confirmed that pizza(especially cheddar and meat pizza) contains sodium, which is known to have a damaging effect on the body.
French fries
Only one portion of French fries has up to 270mg of sodium and 19 grams of fat.
The excessive intake of sodium can lead to water retention and weight gain.
Ramen noodles
Only one bundle of Ramen noodles has 14 grams of fat and 1580 mg of sodium.
Frozen pot pies
One sweet pie contains 1300-1400mg of sodium and 35 g of fat. This is more than the recommended and allowed daily limit.
Canned biscuits
Canned biscuits are packed with processed substances and are excessively seasoned. When you are buying biscuits, make sure to check the sodium content and the ingredients listed on the label.
Canned chicken noodle soup
Canned chicken noodle soup contains excessive amounts of sodium. In fact, only 1 serving of this soup has 800 mg of sodium.
Pickles
Considering their low-calorie content, you would never imagine that pickles are high in sodium. But, a medium-sized pickle contains 570 mg of sodium, which is 1/3 more of the recommended daily intake.
 
Margarine
Even though margarine is a common everyday used ingredients, the reality is that it is one of the unhealthiest ingredients. It is packed with nickel, platinum, and aluminum.
Red meat
It is very important to try to avoid junk food and trans-fat based foods like red meat as much as possible because they can have a damaging effect on your heart and veins.
Sugar
Sugar, especially the artificial one, is linked to serious heart problems and hypertension.
Doughnuts
Even though they are delicious, doughnuts are abundant with calories and fat. Actually, only one doughnut has 200 calories and 12 grams of fat.
Processed meat
In most cases, the excessive consumption of bologna, bacon, frankfurter, and wieners leads to serious heart issues. So, in order to keep your heart healthy and avoid high blood pressure, make sure to stay away from processed meats. It is recommended to eat low-salt meat like chicken, turkey bosom, and incline hamburger.
Liquor
According to many studies the alcohol can reduce blood supply levels. A study conducted in South Korea showed that liquor is one of the main reasons for hypertension.
Pop
The America Heart Association conducted a study which confirmed that sodas and contain artificial sugar, which is known to be the main cause of hypertension.
Foods which help to reduce blood pressure
Fiber-dense foods can help you to reduce the blood pressure. Make sure to consume pasta, rice, grain oats, peas, veggies, and beans. They are abundant with minerals and vitamins and they will keep you full for a longer period of time
Natural products and veggies
Foods which are high in potassium will help to lower sodium levels, thus prevent hypertension. You should consume more tomatoes, oranges, bananas, spinach, lima beans, and sweet potatoes.
Herbs and spices
Do not make it a practice of yours to add more salt to the dishes. Instead, choose healthy herbs and recommended spices to enrich the meals. Try adding basil along with mixed greens. The same goes for thyme, rosemary, sage and oregano.
Sources:
www.goodmorningcenter.com
thescienceofeating.comwww.justamazingrecipes.com

Post Polio Litaff, Association A.C _APPLAC Mexico

Apr 26, 2017

Man moves paralyzed legs using device that stimulates spinal cord








ROCHESTER, Minn. – Mayo Clinic researchers used electrical stimulation on the spinal cord and intense physical therapy to help a man intentionally move his paralyzed legs, stand and make steplike motions for the first time in three years.
The case, the result of collaboration with UCLA researchers, appears today in Mayo Clinic Proceedings. Researchers say these results offer further evidence that a combination of this technology and rehabilitation may help patients with spinal cord injuries regain control over previously paralyzed movements, such as steplike actions, balance control and standing.
Journalists: For a profile on Jered Chinnock and more on the research, visit our News Network.
“We’re really excited, because our results went beyond our expectations,” says neurosurgeon Kendall Lee, M.D., Ph.D., principal investigator and director of Mayo Clinic’s Neural Engineering Laboratory. “These are initial findings, but the patient is continuing to make progress.”
The 26-year-old patient injured his spinal cord at the sixth thoracic vertebrae in the middle of his back three years earlier. He was diagnosed with a motor complete spinal cord injury, meaning he could not move or feel anything below the middle of his torso.
The study started with the patient going through 22 weeks of physical therapy. He had three training sessions a week to prepare his muscles for attempting tasks during spinal cord stimulation. He was tested for changes regularly. Some results led researchers to characterize his injury further as discomplete, suggesting dormant connections across his injury may remain.
Following physical therapy, he underwent surgery to implant an electrode in the epidural space near the spinal cord below the injured area. The electrode is connected to a computer-controlled device under the skin in the patient’s abdomen. This device, for which Mayo Clinic received permission from the U.S. Food and Drug Administration for off-label use, sends electrical current to the spinal cord, enabling the patient to create movement.
After a three-week recovery period from surgery, the patient resumed physical therapy with stimulation settings adjusted to enable movements. In the first two weeks, he intentionally was able to:
  • Control his muscles while lying on his side, resulting in leg movements
  • Make steplike motions while lying on his side and standing with partial support
  • Stand independently using his arms on support bars for balance
Intentional, or volitional, movement means the patient’s brain is sending a signal to motor neurons in his spinal cord to move his legs purposefully.
“This has really set the tone for our post-surgical rehabilitation – trying to use that function the patient recovered to drive even more return of abilities,” says Kristin Zhao, Ph.D., co-principal investigator and director of Mayo Clinic’s Assistive and Restorative Technology Laboratory.
The Mayo researchers worked closely with the team of V. Reggie Edgerton, Ph.D., at UCLA on this study, which replicates earlier research done at the University of Louisville. The Mayo study marks the first time a patient intentionally controlled previously paralyzed functions within the first two weeks of stimulation.
The data suggest that people with discomplete spinal cord injuries may be candidates for epidural stimulation therapy. However, more research is needed into how a discomplete injury contributes to recovering function.
Teams from Mayo Clinic’s departments of Neurosurgery and Physical Medicine and Rehabilitation, and the Division of Engineering collaborated on this project.
“While these are early results, it speaks to how Mayo Clinic researchers relentlessly pursue discoveries and innovative solutions that address the unmet needs of patients,” says Gregory Gores, M.D., executive dean of research, Mayo Clinic.  “These teams highlight Mayo Clinic’s unique culture of collaboration, which brings together scientists and physician experts who work side by side to accelerate scientific discoveries into critical advances for patient care.”
Co-authors are:
  • Peter Grahn, Ph.D., Mayo Clinic
  • Igor Lavrov, M.D., Ph.D., Mayo Clinic
  • Dimitry Sayenko, Ph.D., UCLA
  • Meegan Van Straaten, Mayo Clinic
  • Megan Gill, Mayo Clinic
  • Jeffrey Strommen, M.D., Mayo Clinic
  • Jonathan Calvert, Mayo Clinic
  • Dina Drubach, Mayo Clinic
  • Lisa Beck, Mayo Clinic
  • Margaux Linde, Mayo Clinic
  • Andrew Thoreson, Mayo Clinic
  • Cesar Lopez, Mayo Clinic
  • Aldo Mendez, M.D., Mayo Clinic
  • Parag Gad, Ph.D., UCLA
  • Yury Gerasimenko, Ph.D., UCLA
The research was funded by Craig H. Neilsen Foundation, Jack Jablonski BEL13VE in Miracles Foundation, Mayo Clinic Center for Clinical and Translational Sciences, Mayo Clinic Rehabilitation Medicine Research Center, Mayo Clinic Transform the Practice, and The Grainger Foundation.
The Broccoli Foundation and Christopher and Dana Reeve Foundation supported the UCLA team.
Drs. Edgerton, Gerasimenko and Gad have a financial interest in NeuroRecovery Technologies.
About Mayo Clinic ProceedingsMayo Clinic Proceedings is a monthly peer-reviewed medical journal that publishes original articles and reviews dealing with clinical and laboratory medicine, clinical research, basic science research, and clinical epidemiology. Mayo Clinic Proceedings is sponsored by Mayo Foundation for Medical Education and Research as part of its commitment to physician education. It publishes submissions from authors worldwide. The journal has been published for more than 80 years and has a circulation of 130,000. Articles are at mayoclinicproceedings.org.
About Mayo ClinicMayo Clinic is a nonprofit organization committed to clinical practice, education and research, providing expert, whole-person care to everyone who needs healing. For more information, visit mayoclinic.org/about-mayo-clinic or newsnetwork.mayoclinic.org.
MEDIA CONTACTSSusan Barber Lindquist or Rhoda Madson, Mayo Clinic Public Affairs, 507-284-5005, newsbureau@mayo.edu

Post Polio Litaff, Association A.C _APPLAC Mexico

Apr 24, 2017

For 60 Years, This Woman Defied Her Medical Diagnosis Living INSIDE An Iron Lung



One might think that every bad thing that happens in life is insurmountable. This woman makes us reflect: there’s no greater gift than life itself and she has been able to enjoy it for 71 years.
Martha Mason was a girl like any other in the 1940s. She lived happily together with her family in North Carolina, USA, until one day, when she was diagnosed with polio.
Martha’s brother had died because of the illness, so, to avoid making her parents even more depressed, Martha decided to hide it from them.
It wasn’t long, however, before her parents realized she was suffering from the same disease.
viralstories.tv
viralstories.tv
At just 11 years old, Martha took a decision that would change her life. What she didn’t know is that she would go on to live a further 60 years, because surprisingly, she was able to outlast the one year left to live prognosis given to her by doctors.
Due to her illness, she was left totally paralyzed from the neck down. Martha chose to live in an iron lung instead of living with tubes attached to her. This lung became one of her most faithful allies. Thanks to this metal box that was 6 and a half feet long and weighed 1,800 pounds, she was able to breathe.

Daily Mail
For years, Martha received the support of her family and school friends. Her condition didn’t stop her graduating with honors from college and even becoming the author of a local paper.
In the mid 1990s, Martha Mason decided to write a book called Breath: A Lifetime in
the Rhythm of an Iron Lung, telling her story with the help of a voice-activated computer.

viralstories.tv
She wrote that she never chose to live in such a way, but that she was happy to have so many people supporting her.
In 2009, Martha died at 71 years of age. She is known for having the record of having lived the longest amount of time inside an artificial lung. Her doctor believes that she was able to do it because of her strength and her overwhelming desire to continue living and learning.
This story shows us that, despite the conditions or difficulties we are faced with, we must always fight to overcome them.
Post Polio Litaff, Association A.C _APPLAC Mexico

Apr 22, 2017

Becky, Barbie's friend who uses a wheelchair, was discontinued.



Last year, Mattel announced that it was giving Barbie a makeover — introducing new body shapes, skin tones and even flat feet to make the iconic doll look more realistic. “Barbie reflects the world girls see around them,” Mattel president and COO Richard Dickson said.
Here's why. Studio 360 April 09, 2017 · 10:30 AM EDT Writer Julia FranzPlayer utilities
00:00
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This story is based on a radio interview. Listen to the full interview.
Mattel made a similar statement in 1997 when it introduced “Share-a-Smile Becky,” a Barbie friend who used a wheelchair — but that time, the toymaker’s efforts didn’t go quite as planned.
With her shiny pink wheelchair and tiny backpack, Becky was an instant hit. As many as 6,000 dolls were sold in the first two weeks, and disability advocates praised Mattel for bringing visibility and representation to wheelchair users.
But the warm, fuzzy feeling didn’t last. Kids and collectors soon discovered that Becky’s wheelchair didn’t fit through the doors of the Barbie Dreamhouse — that pink-swathed epicenter of Barbie’s social world. The chair couldn’t squeeze inside the house’s elevator, either.
At the time, Mattel responded to the controversy by saying that the company was “looking at the accessibility of all Barbie accessories.” But as producer Renee Gross reports, that’s not the end of Becky’s lesson about disability and inclusivity. 
Recently, Gross brought a “Share-a-Smile Becky” doll and a brand-new Barbie Dreamhouse to Monique Kulick, an accessibility advocate based in Ann Arbor, Michigan. As it turns out, Becky’s wheelchair still doesn’t fit in the Dream House elevator, 20 years later.
“There's absolutely no way,” Kulick says. “It won't even fit with her legs sticking out. So pretty much in this house, Becky could go to the kitchen.”
And even then, she says, there would still be problems. “If this were Becky's house, her kitchen would have a clear space under the sink so she could roll directly up to it. Dishwashers are normally for an accessible kitchen and universal. They're raised, so that that bottom rack, you don't have to bend down so far for the bottom rack.”
The bottom line? Mattel never changed the house, Gross says. “But what did change was Becky.”
She explains that "Share a Smile Becky" became "Becky, I'm the School Photographer," then "Sign Language 'I Love You' Becky" and then "Paralympic Becky."
Finally, Becky disappeared from shelves altogether — but not before Mattel reportedly considered making her wheelchair smaller, so she could fit through the doors of Barbie’s house.
“A lot of the talk about why Becky doesn't exist anymore in any iteration is that it was too complicated to redesign Barbie world to fit Becky,” says Karin Hitselberger, who has blogged about the Becky dolls she had as a child. “So, they just got rid of her.”
For Hitselberger, who has cerebral palsy and uses a wheelchair, Becky’s story “speaks volumes to the way we think about disability.”
“A lot of the ways we think about disabilities, we talk about ‘fixing disability,’ instead of focusing on ‘fixing society,’” she says.
Kulick agrees. “Oh my god, that’s a huge thing — a huge thing,” she says.
This article is based on a story that aired on PRI's Studio 360 with Kurt Andersen. 


Post Polio Litaff, Association A.C _APPLAC Mexico

Curbing Damage New small molecule treatments could reduce damage due to diabetic eye disease




A team led by Harvard Medical School researchers at Massachusetts Eye and Ear has identified a new therapeutic target for abnormal blood vessel growth in the retina, a hallmark of advanced diabetic eye diseases such as diabetic retinopathy.
According to a report published online in Diabetes on April 11, the transcription factor RUNX1 was found in abnormal retinal blood vessels, and by inhibiting RUNX1 with a small molecule drug, the researchers achieved a 50 percent reduction of retinopathy in preclinical models. These findings pave the way for new therapies that address diabetic retinopathy and other conditions involving abnormal vessel growth within the retina, known as retinal neovascularization.
“We’re hopeful that we may have an opportunity to change the treatment paradigm for these conditions.” —Leo Kim
“Current treatments to control retinal neovascularization require injecting very large proteins, including antibodies, into the eyes of patients, as often as once a month. Our study opens the door for new modalities of treatment based on small molecules that could cross biological barriers on their own. Such a treatment could be self-administered by patients and eliminate the need for intravitreal injections,” said co-corresponding author Joseph Arboleda-Velasquez, HMS assistant professor of ophthalmology and assistant scientist at Schepens Eye Research Institute of Mass. Eye and Ear.
Neovascularization is a feature of various health conditions, including diabetic retinopathy, wet age-related macular degeneration, retinopathy of prematurity and cancer. In the case of diabetic retinopathy—the most common diabetic eye disease and a leading cause of blindness in American adults—blood vessels in the retina (the structure in the back of the eye that senses and perceives light) become damaged and leak fluid. Accumulation of fluid into the retina can lead to swelling at the center of the retina and growth of pathological blood vessels on its surface. As diabetes-related damage progresses, these vessels can leak, rupture or cause retinal detachment leading to impaired vision. 
In the Diabetes report, the authors studied tissue from patients with proliferative diabetic retinopathy. They identified the presence of RUNX1 in the diseased blood vessels but not in the normal blood vessels. Next, they used a small molecule drug originally developed as a cancer therapy to inhibit the activity of RUNX1 in the eye, which led to a significant reduction of abnormal blood vessels.
Current strategies for treating abnormal blood vessel growth in the retina for proliferative diabetic retinopathy include laser treatments or eye injections targeting a growth factor, VEGF. While these therapies have been remarkably successful in saving vision in many patients, they can, in rare instances, trigger complications such as retinal hemorrhages, detachments or retinal atrophy.
The study authors are hopeful that inhibiting RUNX1 may present a more targeted opportunity for managing the retinopathy of certain eye conditions—perhaps earlier in the disease process, before the abnormal blood vessels develop. Future studies will test whether the drug can be delivered through topical eye drops rather than by injection and further explore the relationship between RUNX1 and VEGF, as these factors seemingly both play a role in angiogenesis.
“We’re hopeful that we may have an opportunity to change the treatment paradigm for these conditions,” said co-corresponding author Leo Kim, HMS assistant professor of ophthalmology and a retina surgeon at Mass. Eye and Ear. “Instead of treating patients after these abnormal blood vessels form in the eye, we may be able to give patients eye drops or systemic medications that prevent their development in the first place.”
Adapted from a Mass. Eye and Ear news release.
This study was supported by the National Institutes of Health (grants R01EY005318, R00EY021624, UH2NS100121-01, R21EY027061, K12EY16335 and P30EY003790). Additional support was provided by an American Diabetes Association Innovation award, the Massachusetts Lions Eye Research Fund, the E. Matilda Ziegler Foundation for the Blind, the Karl Kirchgessne

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